Nitro Redox Medicine- Medicine of the future for chronic
Degenerative Disease Prevention
Mitochondrial function, cellular signaling, cellular proliferation, differentiation and apoptosis, immune modulation, cancer growth, cardiac rhythm and circadian rhythm, neuroplasticity, sympathetic tone, enzymatic function, receptor function, endothelial function, gene expression and telomere length are all examples of Redox mediated processes supported in peer-reviewed literature.
Our body’s antioxidant defense mechanisms represent an active process fulfilled by metabolic energy systems, antioxidant enzymes and epigenetics, in addition to the roles of more well-known small molecule antioxidants and related detox compounds.
Reactive oxygen species (ROS) are necessary participants in normal biochemical functions but can induce pathology when imbalanced. The endogenous production of ROS during normal signaling is essential for cellular regeneration and healing effects.
However, excessive ROS production and accumulation lead to detrimental processes that cause degeneration and cellular injury. Oxidative stress-related disease occurs when antioxidant defenses cannot balance the production of ROS.
Redox manipulation can improve homeostasis and cellular health, but it can also be used as a clinical tool. We are just now seeing revolutionary new therapies being developed from this concept.
The age-dependent Nitro-redox imbalance is closely related to oxidative stress and the various syndromes associated with “aging”. ROS overproduction is involved in diverse oxidative stress-related diseases such as degenerative, autoimmune, metabolic, chronic inflammatory, chronic infectious, and psychological diseases, plus cancers, aging, pain, and many more.
In order to develop effective therapeutic approaches against oxidative stress-related diseases, it is important to identify the mechanisms underlying the initial pathological events. Targeted antioxidant treatments are a promising therapeutic approach.
However, the complex nature of oxidative stress-related diseases and the failure of monotherapies suggests that an antioxidant therapy should be accompanied by additional targeted therapeutic interventions to enhance the restoration of the Nitro-redox balance. This is what we provide at EHC BUFFALO.
